Lyme disease manifests in three stages: early, early disseminated, and late stage.
Lyme borreliosis is a complex multi-system vector-borne disease that presents in a multitude of ways. Thirty different pathogens carried by the source can also infect the patient, which further complicates diagnosis. The four most common coinfections: Bartonella, Ehrlichia, Rocky Mountain spotted fever and Babesia.
Depending on a patient’s stage, lab results, and commitment to treatment, it can take a family a year or five years to achieve remission. In the grand scheme of life, that’s very little time to gain a whole new lease on life.
A patient can live relatively symptom-free as long as they are committed to their new lifestyle.
Early – 1st Lyme Disease Stage
The first phase of Lyme disease is known as the early stage. If treated within the first month, the infected person can usually overcome it with a prescription antibiotic. Treatment in the first 30 days can also mean the difference between rapid recovery and a chronic condition.
Symptoms begin three to thirty days after contracting the disease. Initial symptoms include a lack of energy, headaches and a stiff neck, fever and chills, muscle and joint pain, and swollen lymph nodes. These generic symptoms may be misdiagnosed, especially if the patient was unaware of the initial bite or contraction of Lyme disease.
Some patients do not experience any symptoms initially. Less than 50 percent of those infected see an expanding, circular red “bull’s eye” rash before seeing a doctor. The rash is medically known as erythema chronicum migrans, Latin for “chronic migrating redness.” It may be warm to the touch, but it does not itch and is not painful.
The infection rash is a sign that the bacteria are multiplying in the bloodstream. The bullseye can last up to four weeks. Some people will have a solid red rash. On darker-skinned people, the rash may look like a bruise. Therefore, you need to be aware of the other early-stage symptoms.
Other symptoms to look for are a rash on any part of the body, loss of facial muscle use, conjunctivitis, and swelling of the joints. An infected person may feel flu-like symptoms, abnormal pain, weakness or numbness in the arms or legs, headaches, fainting, reduced concentration or brain fog, damage to deep eye tissue, passing pain, heart palpitations, and in rare cases, serious heart problems.
Early Disseminated – 2nd Lyme Disease Stage
The second phase of Lyme disease typically lasts until the sixth month after the initial infection. As the disease progresses, the infected person will generally feel slightly sick for an extended period. They will have the sense they are unable to “shake it off.” This disease invades multiple bodily systems like the nervous system, immune system, and endocrine system. The infected person will become aware that something is wrong and potentially be overwhelmed or crippled by the attacks.
The early-stage symptoms will possibly begin to worsen or multiply. Studies show that bacterial spirochetes actively infect the central nervous system. A variety of neurological issues also start to manifest as the disease progresses to the final stage-chronic stage. The patient may suffer from memory lapses, mood swings, sleep disruption, learning impairment, or difficulty with decision-making. Their knees may develop arthritis or recurring episodes of swelling, redness, and fluid buildup.
Late Stage – 3rd Lyme Disease Stage
The final stage of Lyme disease is when it becomes a chronic condition. The symptoms experienced in the previous stages may continue, worsen, or alleviate. Less than 20 percent of those infected with Lyme enter the late stage or chronic phase.
Numbness or tingling in the hands, feet, or back may also occur. In rare cases, heart problems may begin. The most severe heart problem is pericarditis, the inflammation of the structures surrounding the heart.
As the disease progresses, the body may experience a variety of changes. Neurotoxins could change the chemical signals within the body. Typically, there is a disruption in the pineal gland, hypothalamus, and pituitary gland. Which may present as an enzyme deficiency or inhibition, significant metabolic changes (KryptoPyrole Urea, aka “KPU”), emotional and psychiatric presentation, neurotransmitter depletion, hormone imbalance, and a synergistic effect with heavy metals, xenobiotics, thioethers, root-filled teeth, and more.
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